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Fredriksson L.'s CAN for critical embedded automotive networks PDF

By Fredriksson L.

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Garg M, Devaskar SU: Glucose metabolism in the late preterm infant. Clin Perinatol 2006;33:853 [PMID: 17148009]. ᮢ THE NEWBORN INFANT 19 Hussain K, Aynsley-Green A: The effect of prematurity and intrauterine growth restriction on glucose metabolism in the newborn. NeoReviews 2004;5:e365. Kahler SG: Metabolic disorders associated with neonatal hypoglycemia. NeoReviews 2004;5:e377. Nold JL, Georgieff MK: Infants of diabetic mothers. Pediatr Clin North Am 2004;51:619 [PMID: 15157588]. Rozance PJ, Hay W: Hypoglycemia in newborn infants: Features associated with adverse outcomes.

Liver transplantation is curative (see Chapter 21). 2. Gilbert syndrome—This is a mild autosomal dominant disorder characterized by decreased hepatic UDPGT activity caused by genetic polymorphism at the promoter region of the UDPGT gene. 3. Affected individuals tend to develop hyperbilirubinemia in the presence of conditions that increase bilirubin load. They are also more likely to have prolonged neonatal jaundice and breast milk jaundice. C. Hyperbilirubinemia Caused by Unknown or Multiple Factors 1.

Hemolysis is usually mild, but the severity is unpredictable because of variability in the amount of naturally occurring maternal anti-A or anti-B IgG antibodies. Although 20% of pregnancies are “set-ups” for ABO incompatibility (mother O, infant A or B), only 33% of infants in such cases have a positive direct Coombs test and only 20% of these develop jaundice that requires therapy. Persistent maternal antibodies may cause the newborn to gradually become anemic over the first few weeks of life, even to the point of requiring transfusion.

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